By Christopher Loftus
This version of Neurosurgical Emergencies is an up to date reference overlaying the main often encountered neurosurgical emergencies brought on by ailment, an infection ...
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Additional resources for Neurosurgical emergencies
During the last several decades, numerous investigations and researches have improved our understanding of the pathophysiology of intracranial hypertension. In addition, the availability of advanced neuroimage and monitoring technologies has resulted in effective management for the patient with central nervous system diseases associated with intracranial hypertension. Uncontrollable intracranial hypertension has been shown to be the primary cause of death in most patients who die of central nervous system diseases such as traumatic brain injury (TBI) and stroke.
In: Plum F, Posner JB. The Diagnosis of Stupor and Coma. 3rd ed. Philadelphia, PA: FA Davis;1982:87–151 4. Cairns H. Disturbances of consciousness with lesions of the brainstem and diencephalon. Brain1952;75(2):109–146 5. Plum F, Posner JB. The pathologic physiology of signs and symptoms of coma. In: Plum F, Posner JB. The Diagnosis of Stupor and Coma. 3rd ed. Philadelphia, PA: FA Davis; 1982:1–86 6. Moulton R. Head injury. In: Young GB, Ropper AH, Bolton CF, eds. Coma and Impaired Consciousness.
Thiamine 100 mg IV would prevent precipitation of Wernicke encephalopathy by glucose infusion in malnourished or alcoholic patients. Antidotes for narcotic (naloxone) or benzodiazepine (flumazenil) overdosage only awaken the patient temporarily; although this serves a diagnostic purpose, convulsions may occur with flumazenil. Bloodwork for electrolytes, glucose, renal and hepatic function, calcium, creatine kinase, ammonia, thyroid stimulating hormone (TSH), blood count, protime and activated partial thromboplastin time (aPTT) should be sent, in addition to an arterial blood gas (with carbon monoxide level if suspected), and urine drug screen.